Production of Blister in Normal Human Skin In Vitro By Blister Fluids from Epidermolysis Bullosa

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T cells are required for the production of blister-inducing autoantibodies in experimental epidermolysis bullosa acquisita.

Epidermolysis bullosa acquisita is a prototypical organ-specific autoimmune disease caused by autoantibodies against type VII collagen of the dermal-epidermal junction. Although mechanisms of autoantibody-induced blister formation were extensively characterized, the initiation of autoantibody production in autoimmune blistering diseases is still poorly defined. In the current study, we addresse...

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The alternative pathway of complement activation is critical for blister induction in experimental epidermolysis bullosa acquisita.

Epidermolysis bullosa acquisita is a subepidermal blistering disease associated with tissue-bound and circulating autoantibodies against type VII collagen, a major constituent of the dermal-epidermal junction. The passive transfer of Abs against type VII collagen into mice induces a subepidermal blistering disease dependent upon activation of terminal complement components. To further dissect t...

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ژورنال

عنوان ژورنال: Journal of Investigative Dermatology

سال: 1984

ISSN: 0022-202X

DOI: 10.1111/1523-1747.ep12260378